RT Journal
A1 Wiley HE, III, Eaglstein WE
T1 CAlcinosis cutis in children following electroencephalography
JF JAMA
JO JAMA
YR 1979
FD August 3
VO 242
IS 5
SP 455
OP 456
DO 10.1001/jama.1979.03300050045027
UL http://dx.doi.org/10.1001/jama.1979.03300050045027
AB WE RECENTLY cared for seven children in whom calcinosis cutis developed at the sites of EEG electrode placement. The lesion apparently is produced by penetration of calcium chloride from the EEG paste into deliberately abraded skin and may be prevented by slight alterations in techniques.Report of a Case 
    A premature boy was transferred to a neonatal intensive care unit shortly after birth because of respiratory distress, jaundice, hepatosplenomegaly, thrombocytopenia, and an elevated IgM level. His course was complicated by pneumonia, congestive heart failure secondary to cor pulmonale, and repeated apneic episodes. His EEGs at 3 and 5 months of age were normal.Shortly after the second EEG, skin lesions were noted in the distribution of the previously applied EEG leads on his scalp and earlobes (Fig 1). Each lesion was a discrete, approximately 1-cm red papule surmounted by multiple yellowish milia. A biopsy specimen of one lesion showed a