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Screening and Detection of Breast Cancer and Prostate Cancer

Emmanuel Saloustros, MD; Dimitris Mavroudis, MD
JAMA. 2010;303(11):1032-1034. doi:10.1001/jama.2010.273
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To the Editor: The Special Communication by Dr Esserman and colleagues1 summarized several observations after 20 years of extensive screening for breast and prostate cancer (1986-2006). The authors referred to data from the Surveillance, Epidemiology and End Results (SEER) program to report that the incidence of total invasive breast cancer (excluding in situ lesions) has increased substantially and remains higher than prescreening rates, while the localized (node negative, no skin or chest wall involvement) and regional (node positive, skin or chest wall involvement) breast cancer incidence has declined only slightly. The authors concluded that the increase in the relative fraction of early stage cancers without decrease in the incidence of regional cancers is the result of preferential detection of the slower growing and potentially indolent tumors (length bias).

Although we share the authors' concern, it is important to note that data collected by the SEER program between 1992 and 1998 suggested a difference in the incidence of breast cancer according to hormonal receptor status. The incidence of estrogen receptor– (ER) and/or progesterone receptor– (PR) negative breast cancer was declining while that of ER/PR-positive disease was increasing.2 At least some of this increase might be attributable to the use of hormone therapy in the 1980s and 1990s, since hormone therapy may preferentially increase the risk of ER/PR-positive tumors. Furthermore, hormone-positive tumors are more likely to be diagnosed in less advanced stages than the tumors without expression of hormonal receptors.3

Therefore, it is possible that the localized tumors detected by screening mammography represent, at least in part, cancers attributed to hormonal therapy. It is hoped that the increasing use of screening mammography resulted in early diagnosis, before locally advanced disease or metastatic spread developed. Alternatively, some cancers would have been diagnosed at advanced stages, resulting in a similar increase in the incidence of late stage breast cancer compared with early stage disease. The decrease in the incidence of 3.5% per year from 2001 to 2004 with little additional decrease in 20044 (coincident with the decreased use of hormone therapy because of the data from the Women's Health Initiative linking hormone therapy and breast cancer) supports this hypotheses.5 The differences were unrelated to changes in the frequency of screening mammography.

AUTHOR INFORMATION

Financial Disclosures: None reported.

REFERENCES

Esserman L, Shieh Y, Thompson I. Rethinking screening for breast cancer and prostate cancer.  JAMA. 2009;302(15):1685-1692
PubMedCrossRef
Li CI, Daling JR, Malone KE,  et al.  Incidence of invasive breast cancer by hormone receptor status from 1992 to 1998.  J Clin Oncol. 2003;21(1):28-34
PubMedCrossRef
Tinnemans JG, Beex LV, Wobbes T, Sluis RF, Raemaekers JM, Benraad T. Steroid-hormone receptors in nonpalpable and more advanced stages of breast cancer: a contribution to the biology and natural history of carcinoma of the female breast.  Cancer. 1990;66(6):1165-1167
PubMedCrossRef
Jemal A, Siegel R, Ward E, Hao Y, Xu J, Thun MJ. Cancer statistics, 2009.  CA Cancer J Clin. 2009;59(4):225-249
PubMedCrossRef
Chlebowski RT, Kuller LH, Prentice RL,  et al; WHI Investigators.  Breast cancer after use of estrogen plus progestin in postmenopausal women.  N Engl J Med. 2009;360(6):573-587
PubMedCrossRef

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Esserman L, Shieh Y, Thompson I. Rethinking screening for breast cancer and prostate cancer.  JAMA. 2009;302(15):1685-1692
PubMedCrossRef
Li CI, Daling JR, Malone KE,  et al.  Incidence of invasive breast cancer by hormone receptor status from 1992 to 1998.  J Clin Oncol. 2003;21(1):28-34
PubMedCrossRef
Tinnemans JG, Beex LV, Wobbes T, Sluis RF, Raemaekers JM, Benraad T. Steroid-hormone receptors in nonpalpable and more advanced stages of breast cancer: a contribution to the biology and natural history of carcinoma of the female breast.  Cancer. 1990;66(6):1165-1167
PubMedCrossRef
Jemal A, Siegel R, Ward E, Hao Y, Xu J, Thun MJ. Cancer statistics, 2009.  CA Cancer J Clin. 2009;59(4):225-249
PubMedCrossRef
Chlebowski RT, Kuller LH, Prentice RL,  et al; WHI Investigators.  Breast cancer after use of estrogen plus progestin in postmenopausal women.  N Engl J Med. 2009;360(6):573-587
PubMedCrossRef
March 17, 2010
Nigel Paneth, MD, MPH; George Vande Woude, PhD; Eric Kort, MD, MSc
JAMA. 2010;303(11):1032-1034.
March 17, 2010
Marc B. Garnick, MD
JAMA. 2010;303(11):1032-1034.
March 17, 2010
Laura Esserman, MD, MBA; Ian Thompson, MD
JAMA. 2010;303(11):1032-1034.
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