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Risk of Venous Thromboembolism With Bevacizumab in Cancer Patients

David R. Minor, MD
JAMA. 2009;301(14):1434-1436. doi:10.1001/jama.2009.439
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Published online

To the Editor: The meta-analysis assessing the risk of venous thromboembolism (VTE) with bevacizumab in cancer patients by Dr Nalluri and colleagues1 carefully reviewed 15 well-conducted randomized controlled trials (RCTs) comparing bevacizumab therapy with placebo. However, because the authors failed to distinguish between incidence per treatment episode and incidence per unit of time (the more relevant statistic), their conclusions may be erroneous. In oncology clinical trials such as those reviewed, patients are generally not treated for a predetermined fixed period of time; rather, they are treated until their cancer shows evidence of progression on computed tomographic scans, at which time the patient is removed from the trial and further adverse events are not recorded beyond 30 days after the end of treatment.

Because bevacizumab is a beneficial oncology drug, the patients in these trials often stayed on treatment with bevacizumab much longer than placebo, giving them more time to develop adverse events such as venous thromboembolism. For example, in the study by Hurwitz et al,2 the median progression-free survival with bevacizumab was 10.6 months vs 6.2 months with placebo. Because time on treatment is very close to time to progression, this is a 70% increase in time available for an adverse event to occur. For Miller et al,3 the increase was 100%, and for Sandler et al,4 37%. Nalluri et al relied on the published data that give crude incidence of thromboembolism per treatment episode, not incidence per unit of time. Given the considerably longer period on treatment for patients receiving bevacizumab, it is no surprise that 33% more venous thromboemboli were recorded in the groups receiving bevacizumab. At the minimum, the data should have been corrected for the published figures for median progression-free survival.

AUTHOR INFORMATION

Financial Disclosures: Dr Minor reported receiving research funds from Genentech as an investigator in a multicenter trial.

REFERENCES

Nalluri SR, Chu D, Keresztes R, Zhu X, Wu S. Risk of venous thromboembolism with the angiogenesis inhibitor bevacizumab in cancer patients: a meta-analysis.  JAMA. 2008;300(19):2277-2285
PubMedCrossRef
Hurwitz H, Fehrenbacher L, Novotny W,  et al.  Bevacizumab plus irinotecan, fluorouracil, and leucovorin for metastatic colorectal cancer.  N Engl J Med. 2004;350(23):2335-2342
PubMedCrossRef
Miller K, Wang M, Gralow J,  et al.  Paclitaxel plus bevacizumab versus paclitaxel alone for metastatic breast cancer.  N Engl J Med. 357;2007(26):2666-2676
PubMed
Sandler A, Gray R, Perry MC,  et al.  Paclitaxel-carboplatin alone or with bevacizumab for non-small-cell lung cancer.  N Engl J Med. 2006;355(24):2542-2550
PubMedCrossRef

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Nalluri SR, Chu D, Keresztes R, Zhu X, Wu S. Risk of venous thromboembolism with the angiogenesis inhibitor bevacizumab in cancer patients: a meta-analysis.  JAMA. 2008;300(19):2277-2285
PubMedCrossRef
Hurwitz H, Fehrenbacher L, Novotny W,  et al.  Bevacizumab plus irinotecan, fluorouracil, and leucovorin for metastatic colorectal cancer.  N Engl J Med. 2004;350(23):2335-2342
PubMedCrossRef
Miller K, Wang M, Gralow J,  et al.  Paclitaxel plus bevacizumab versus paclitaxel alone for metastatic breast cancer.  N Engl J Med. 357;2007(26):2666-2676
PubMed
Sandler A, Gray R, Perry MC,  et al.  Paclitaxel-carboplatin alone or with bevacizumab for non-small-cell lung cancer.  N Engl J Med. 2006;355(24):2542-2550
PubMedCrossRef
April 8, 2009
Javier Cortes, MD, PhD; Cristina Saura, MD; Francesco Atzori, MD
JAMA. 2009;301(14):1434-1436.
April 8, 2009
Saadettin Kilickap, MD; Cagatay Arslan, MD
JAMA. 2009;301(14):1434-1436.
April 8, 2009
David Chu, MD; Shenhong Wu, MD, PhD
JAMA. 2009;301(14):1434-1436.
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