Torsion of the spermatic cord presents a classical clinical picture with which most physicians are familiar. Frequently, however, a correct diagnosis of acute torsion is not made, and testicular infarction ensues, owing to delay on the part of both patient and physician.
Numerous cases have been recorded with many interesting variations. Torsion has been described as developing in the fetus, in the undescended testicle of the adolescent, and in apparently normal males of any age group. It may be unilateral or bilateral. There may be preceding stress or trauma or no demonstrable precipitating factor.1
Anatomic abnormalities predisposing to torsion have been mentioned by a number of authors; Lowsley and Kirwin2 have collected a list of 10 different structural disorders favoring its development. The evident relationship of the condition to anatomic abnormalities might suggest the possibility of familial predisposition; however, until this report no such instance has been recorded