Current research on the causes of arteriosclerosis and coronary heart disease at least encourages an optimistic outlook. Recent findings indicate that these conditions may be acquired diseases rather than inevitable consequences of the aging process. Considerable evidence is being accumulated that these conditions, which are most common in some of the more highly civilized populations and appear to be increasing in prevalence, may be in large measure a result of faulty nutrition. A spirit of hopefulness has been aroused that this scourge may be prevented or checked by appropriate regulation of the diet.
It is imperative that the urgency in the situation not be allowed to disrupt a sound and orderly attack on the problem, through exploitation by selfish interests or undue emotion and competitiveness among investigators. A prime need is for conservative, deliberate, and cautious interpretation and application of the fragments of knowledge seemingly related to the solution of