Scoliosis is today as much a problem as ever. No solution offered so far has stood the test of time. Various therapeutic measures have met with enthusiastic approval, only to be discarded later as inadequate and far below the original expectations. Nevertheless, certain facts have gradually been established, some of which pertain to the so-called adolescent or static scoliosis.
The rôle that the muscles play in the etiology and treatment received some consideration from the pioneers (Haglund and Lorenz1). They understood the changes in function as well as the altered contour of the back. Structural weakness frequently has been found associated with functional changes in scoliosis.2 Curvatures have been found to arise as the result of inadequate functional capacity of the structures. This functional insufficiency is sometimes referred to as a decompensation.3 The primary rôle in the development of a decompensation of adolescent scoliosis is played by