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AN INTERPRETATION OF SOME INTESTINAL FACTORS IN EXPERIMENTAL SCURVY

JAMA. 1918;70(4):235-236. doi:10.1001/jama.1918.02600040033011.
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We recently called attention to the changing point of view which several prominent investigators of that form of experimental scurvy which most closely resembles human scorbutus are taking toward the etiology of the disease.1 The theory that scurvy is a representative "deficiency disease" caused by the lack of suitable "vitamins" or other so-called accessory food substances in the diet—a view originally encouraged by the well tested analogy of vitamin deficiency in the case of beriberi—is being supplanted by considerations of another sort. Thus it is being maintained that the lack of suitable "ballast" in the diet, especially in the case of individuals or species which are adapted to voluminous alimentary contents as an aid to the discharge of feces from the bowel, is a predisposing cause.2 In explanation of the untoward results of a ration devoid of feces-producing factors, it is alleged that impaction of the cecum may

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