Even though sickle cell disease (SCD) is caused by a single-nucleotide mutation (GAT→GTT in the sixth codon of axon 1 of the β globin gene), its clinical presentation involves a multitude of complications involving all organs.1- 3 The manifestations of SCD are recurrent—a painful crisis that is resolved will soon be replaced by a new episode of pain; a leg ulcer that heals may soon return.2
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