Editorial |

Race/Ethnicity, Serum 25-Hydroxyvitamin D, and Heart Disease

Keith C. Norris, MD1,2; Sandra F. Williams, DMD, MD3,4
[+] Author Affiliations
1Geffen School of Medicine, University of California, Los Angeles
2Department of Medicine, Charles R. Drew University, Los Angeles, California
3Department of Endocrinology, Cleveland Clinic, Weston, Florida
4Department of Clinical Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, Florida
JAMA. 2013;310(2):153-155. doi:10.1001/jama.2013.7229.
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A growing body of observational evidence had suggested that low 25-hydroxyvitamin D (25[OH]D) concentrations may be a key biological predictor of increased rates of coronary heart disease (CHD).1 Because low serum 25(OH)D concentrations are more common and more severe among racial/ethnic minority groups, which are also affected by higher rates of CHD and CHD risk factors, low vitamin D was being heralded as a potential modifiable contributor to racial/ethnic disparities in cardiovascular health.2 However, the causal link between 25(OH)D concentrations and CHD remains uncertain.3,4 Additionally, the potential clinical implications of low serum 25(OH)D concentrations, the pathological mechanisms through which vitamin D may modulate CHD, and whether these factors differ across racial/ethnic groups are unclear.

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