A growing body of observational evidence had suggested that low 25-hydroxyvitamin D (25[OH]D)
concentrations may be a key biological predictor of increased rates of coronary heart disease
(CHD).1 Because low serum 25(OH)D concentrations are more
common and more severe among racial/ethnic minority groups, which are also affected by higher rates
of CHD and CHD risk factors, low vitamin D was being heralded as a potential modifiable contributor
to racial/ethnic disparities in cardiovascular health.2
However, the causal link between 25(OH)D concentrations and CHD remains uncertain.3,4 Additionally, the potential clinical implications of
low serum 25(OH)D concentrations, the pathological mechanisms through which vitamin D may modulate
CHD, and whether these factors differ across racial/ethnic groups are unclear.
Some tools below are only available to our subscribers or users with an online account.
Download citation file:
Web of Science® Times Cited: 4
Customize your page view by dragging & repositioning the boxes below.
More Listings atJAMACareerCenter.com >
The Rational Clinical Examination: Evidence-Based Clinical Diagnosis
All results at
Enter your username and email address. We'll send you a link to reset your password.
Enter your username and email address. We'll send instructions on how to reset your password to the email address we have on record.
Athens and Shibboleth are access management services that provide single sign-on to protected resources. They replace the multiple user names and passwords necessary to access subscription-based content with a single user name and password that can be entered once per session. It operates independently of a user's location or IP address. If your institution uses Athens or Shibboleth authentication, please contact your site administrator to receive your user name and password.