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Skaz y lyudini patologichna anatomiy, klinika patogenez, patofiziologiy

JAMA. 1936;107(9):739-740. doi:10.1001/jama.1936.02770350107034.
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This contribution from the Ukrainian Psychoneurologic Academy is based on a study of seventy-five clinical cases of rabies, in seventeen of which a histopathologic study was made. Eight of the latter group were studied by the method of serial sections of the entire cerebrospinal axis. The lesions were focal in character and were found at all levels. Two predominant groups were separated, the inflammatory and the degenerative. In some cases the glial proliferation resembled that of a postvaccination encephalitis and suggested the possible influence of antirabic inoculations. The author disagrees with K. Schaffer, who maintained that most pronounced inflammatory changes were found in the spinal cord segment corresponding to the level of the initial lesion (bite). The alterations of the glial cells were of the grave type described by Nissl. The author disagrees with the opinion of H. Pette and gives support to the view of Spielmeyer that there is


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