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THE PREVENTION OF HYPOCHROMIC ANEMIA IN PREGNANCY

JOHN C. CORRIGAN, M.D.; MAURICE B. STRAUSS, M.D.
JAMA. 1936;106(13):1088-1090. doi:10.1001/jama.1936.02770130038012.
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Within the past decade the high incidence of anemia in pregnancy has become generally appreciated. Although the work of Strauss and Castle,1 Dieckmann and Wegner2 and others has indicated that a 10 to 20 per cent lowering of the hemoglobin during pregnancy may be the result of hydremia and not represent true anemia, approximately 25 per cent of otherwise normal women are definitely anemic following parturition.3 The vast majority of such patients have anemia of the hypochromic variety,4 which has come to be associated with a virtual deficiency of available iron for purposes of blood regeneration within the body. Strauss and Castle,5 Davies and Shelley6 and other clinicians have shown that in pregnancy this anemia is to be associated not only with the presence of the fetus but also with gastric secretory defects and inadequate diets and usually may be completely relieved equally well

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